Good start... what is not obvious, though, is why any two species exhibit the precise degree of genetic similarity that they do, if it were not for the fact that they are related by descent. In other words, if chimpanzees and humans are not related by descent, why do their genomes compare so closely, for example? There is no reason why this should be the case. All species share common genes - genes which encode the same proteins/enzymes that are essential to the most fundamental of life processes - such as cytochrome C. But, these genes (and hence the proteins) are not identical - they are subtley different from species to species... although they actually do the same job.
Qualitatively, the information content and ultimate physical manifestion of these different genes is equivalent - the genes and proteins are different, but the outcome (the function of the protein and the information contained in the DNA in order to make that protein) is the same. So, this being the case, it shouldn't matter which species gets which version of the gene/protein - from a design perspective, there's no reason why cytochrome C proteins should be different at all. Furthermore, there's no reason why cytochrome c proteins should exhibit variable amounts of difference between species either. But they do.
Of all the infinite possible ways that species could have been assigned cytochrome C proteins (and many others besides), only an extremely tiny fraction supports the common descent hypothesis. In contrast, there are literally none that would
not support the intelligent design hypothesis. This is a key point that simply has to be understood - the physical data is just one side of the story. It fits both hypotheses, so both hypotheses are equally good, right? Wrong. The other side of the story is whether or not your hypothesis has any explanatory power or is meaningful in any way. But if all possible observations fit your hypothesis, then the data becomes meaningless, and therefore so must be your hypothesis.
As I have said before in this thread, it is like opening a safe. The observed data is like knowing the combination to the safe - but the usefulness of the safe depends on the number of possible right answers that safe has. If any combination opens your safe, then the safe is lousy - it also doesn't matter what data you have either, since any possible combination will do. But, the predictions made by evolution theory are considerably more specific - that is, if the common descent hypothesis is not to be refuted, then the observed data must fall into that extremely tiny fraction of possible combinations that common descent could generate. In the analogy, this is equivalent to saying that the safe only has a very small number of possible answers that will satisfy it. If the observed data matches, then your hypothesis is good. If it doesn't match, however, then the hypothesis is incorrect.
So, if chimp cytochrome C was as different to human cytochrome C and zebrafish cytochrome C, the hypothesis that chimps and humans are more closely related by descent than humans and zebrafish are would be refuted. Similarly, if chicken and turkey cytochrome C proteins were as different to each other as chicken and lemongrass cytochrome C proteins, again the hypothesis would be refuted. Now extend this argument across the entire living world, and you'll find that there are infinitely more ways for the common descent hypothesis to be refuted than there are for it to be not.
So what does
the actual data say - well, it resoundingly endorses the common descent hypothesis. (Of course, the data doesn't say anything about the intelligent design hypothesis for the reason that I've just explained i.e. the hypothesis cannot be refuted, therefore the data is irrelevant - and when the data is irrelevant, you're not doing science). The fact is, human and chimp cytochrome C proteins are 100% identical, chicken and turkey cytochrome C proteins are also 100% identical
to each other,
but critically, they are not 100% identical to chimp/human cytochrome C's. The pattern of similarities and differences is
not random, nor is it possible that it has occured by chance, therefore there must be a mechanism that is responsible for the observed result. That mechanism was proposed long before the data was available - infact, the mechanism (evolution) was proposed before the existence of genes was
even known.
Evolution theory not only predicted the existence of inheritable biomolecules (genes), but has also correctly predicted the existence of patterns of genetic similarity and difference between species.
This being the case, I refer back to the question I asked before - the same question that no creationist seems willing to answer.
Why, with all the possible ways that life could have been designed, was it designed in a way that makes it look exactly like it evolved? In the context of my point above, why should it be so that the observed data falls into that incredibly unlikely subset that supports the common descent hypothesis, and not any one of a practically infinite number of possible subsets that would soundly refute it?